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Macrophages in chronic type 2 inflammation have a novel phenotype characterized by the abundant expression of Ym1 and Fizz1 that can be partly replicated in vitro

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Furthermore, novel mechanisms such as disulfide stress, disulfidptosis, and protein sulfhydration are discussed, highlighting their diverse roles in cellular regulation and disease progression

Dyer, 2009

PMID: 42009144 Free Radic Biol Med Jejunal epithelial barrier disruption triggered by reactive oxygen species in early SIV infected rhesus macaques